CENSORED: Hydroxychloroquine

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Sen. Ron Johnson (R-Wisconsin) is the latest figure to be censored over providing information about hydroxychloroquine and effectiveness for Covid-19.

Johnson’s account on YouTube, which is owned by Google, was reportedly suspended for seven days.

In response, Johnson tweeted: “YouTube’s arrogant Covid censorship continues. How many lives will be lost as a result? How many lives could have been saved with a free exchange of medical ideas? This suppression of speech should concern every American.”

A great deal of information and misinformation was distributed in the media after then-President Trump said hydroxychloroquine could turn out to be an effective treatment for Covid-19.

Because of the controversy, a major scientific study measuring hydroxychloroquine’s effectiveness as a possible preventive could not be finished, so the question was never answered. However, the independent scientists working on the issue said they believed hydroxychloroquine had shown great promise both as effective early treatment for Covid-19 and as a possible preventive measure.

A new study in MedRxiv found that hydroxychloroquine and zinc increased Covid-19 survival by almost three times.

A recent study published in the Journal of The Association of Physicians of India also found hydroxychloroquine is an effective treatment for Covid-19.

You Tube told Epoch Times that it suspended Johnson’s account and removed a video “…in accordance with our COVID-19 medical misinformation policies, which don’t allow content that encourages people to use Hydroxychloroquine or Ivermectin to treat or prevent the virus.”

Watch my investigation on the politics of hydroxychloroquine here.

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7 thoughts on “CENSORED: Hydroxychloroquine”

  1. Suppression of life saving treatment is unethical. Allowing it to go on for over a year makes you a monster worthy of a war crimes or crimes against humanity charge and conviction.. Unless you are part of the government and corporate cabal that can’t have a treatment for Covid-19 less your emergency use authorization isn’t valid and your fear porn no longer effective. Being the target of psyops is not fun.

  2. Google’s CEO (what’s her name?) should be treated the same as the guilty Nuremberg defendants. My mom likely would still be alive had Google hierarchy not withheld lifesaving information.

  3. And yet the lies continue, and it’s next to impossible to find the facts, ma’am, just the facts with most search engines. Especially google. Not blatantly suspicious at all. Why? Well, denial, obfuscation, diversion and crazy-making are the main tools in the boxes of the current leftist regime. The news (once, but no longer impartial!) and big tech “media” have become governmental arms. With their incredible wealth and “resistance is futile” censorship, they believe they cannot lose. It’s up to the free to resist and keep focusing light upon simple facts.

  4. Abstract
    While vaccination campaigns are ongoing worldwide, there is still a tremendous medical need for efficient antivirals against SARS-CoV-2 infection. Among several drug candidates, chloroquine (CQN) and hydroxychloroquine (H-CQN) were tested intensively, and any contentious therapeutic effect of both has been discussed controversially in the light of severe side effects and missing efficacy. Originally, H-CQN descended from the natural substance quinine, a medicinal product used since the Middle Ages, which actually is regulatory approved for various indications. We hypothesized that quinine also exerts anti-SARS-CoV-2 activity. In Vero cells, quinine inhibited SARS-CoV-2 infection more effectively than CQN, and H-CQN and was less toxic. In human Caco-2 colon epithelial cells as well as the lung cell line A549 stably expressing ACE2 and TMPRSS2, quinine also showed antiviral activity. In consistence with Vero cells, quinine was less toxic in A549 as compared to CQN and H-CQN. Finally, we confirmed our findings in Calu-3 lung cells, expressing ACE2 and TMPRSS2 endogenously. In Calu-3, infections with high titers of SARS-CoV-2 were completely blocked by quinine, CQN, and H-CQN in concentrations above 50 µM. The estimated IC50s were ~25 µM in Calu-3, while overall, the inhibitors exhibit IC50 values between ~3.7 to ~50 µM, dependent on the cell line and multiplicity of infection (MOI). Conclusively, our data indicate that quinine could have the potential of a treatment option for SARS-CoV-2, as the toxicological and pharmacological profile seems more favorable when compared to its progeny drugs H-CQN or CQN.


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